The Health History Of A Student At The University Of Arizona

Introduction This paper will look at the health history of a student at the University of Arizona in the BSN program. This paper will explore the risks this student has of contracting diabetes mellitus due to a predisposition in her family history. The reason for the disease, patterns, risk, and preventative measures will be discussed and explained through the paper. Many people in the world suffer from type II diabetes [diabetes mellitus (DM)]. This student explored her health history back tracing diseases through her sibling, parents, aunts, uncles, and grandparents. This disease has a genetic component, but is also about lifestyle and choices that can be made to prevent the severity of this disease. Pathophysiology Diabetes†¦show more content†¦739). These are all genetic factors that contribute to potentially develop diabetes type II mellitus. These genetic factors alone are not enough to contract diabetes there needs to be environmental factors/life choices. Environmental factors include â€Å"obesity, hypertension, and metabolic syndrome† (McCane, 2014, p. 739). When a person has the environmental â€Å"risk factors for type II diabetes with insulin resistance, only those who have the genetic predisposition to beta cell dysfunction will develop type II diabetes† (McCane, 2014, p. 739). This means that a person who is not considered overweight, but has hypertension or does not live a healthy lifestyle and is predisposed can develop this disease as well. This is a rare occurrence in type II because â€Å"obesity is present in 60-80% of those with type II diabetes† (McCane, 2014, p. 739). Being obese contributes to the development of diabetes in five ways. These five ways are â€Å" 1. Adipokines are hormones produced by adipose tissue. /†¦/ Adipokines have effects not only on tissues but [also] on the hypothalamic and pancreatic function. /†¦/ 2. Elevated serum free fatty acids and high intracellular deposits of triglycerides and cholesterol. These lead to metabolic overload with interfere of insulin signaling and a decrease in tissue response to insulin. 3. Obesity causes release of inflammatory cytokines. /†¦/ These play an important role in the genesis of a fatty liver [which impairs the function]. 4.